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EGF — NPY6R
Text-mined interactions from Literome
Samet et al., Toxicol Appl Pharmacol 2003
:
Exposure to Zn ( 2+ ), but not
EGF , induced phosphorylation of the activating site of c-Src ( tyrosine 416 ), and Zn ( 2+ ) -induced phosphorylation of EGFR at tyrosines 845 and 1068 was
blocked by the c-Src kinase activity inhibitor
PP2
Xu et al., Invest Ophthalmol Vis Sci 2007
:
The release of HB-EGF assessed by AP activity increased significantly in response to wounding, LPA, or both, and the release of
HB-EGF-AP induced by LPA was
inhibited by
PP2 and GM6001
Lin et al., Am J Physiol Cell Physiol 2008
:
The Src inhibitor
PP2 blocked pilocarpine induced ERK activation and
EGF receptor phosphorylation, without affecting ERK activation by carbachol
Suh et al., J Cell Physiol 2009
:
In addition, IL-6 increased EGFR/src/FAK, PI3K/Akt phosphorylation and 2-DG uptake as well as GLUT-2 protein expression, which were blocked by AG 1478 (
EGF receptor
inhibitor ),
PP2 ( src family of tyrosine kinase inhibitor ), PI3K-specific siRNA, and a Akt inhibitor
Dong et al., Biochim Biophys Acta 2011
:
PP2 , a c-Src specific inhibitor,
suppressed both TCDD- and
epidermal growth factor- (EGF) induced c-Src activation