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NOX5 — PPARA

Text-mined interactions from Literome

von Knethen et al., Free Radic Biol Med 2011 : NADPH oxidase activation in either RAW264.7 cells or peritoneal macrophages ( PM ) derived from PPAR? wild-type mice increased reactive oxygen species ( ROS ) formation, caused PPAR? activation, heme oxygenase-1 (HO-1) induction, and concomitant IFN-ß expression
Wagner et al., Alcohol Clin Exp Res 2012 : PPAR? ligands regulate NADPH oxidase , eNOS, and barrier function in the lung following chronic alcohol ingestion
Wu et al., J Endocrinol Invest 2013 : Trx and PPAR? were significantly decreased, and Txnip expression and NADPH oxidase activity markedly increased , in the D and T groups compared to the C group
Green et al., Pulmonary circulation 2012 : We previously demonstrated that hypoxia induced human pulmonary artery smooth muscle ( HPASMC ) cell proliferation and expression of the NADPH oxidase subunit, Nox4, were attenuated by the peroxisome proliferator activated receptor ? ( PPAR? ) agonist, rosiglitazone