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GRAP2 — RAC2

Text-mined interactions from Literome

Philips et al., J Biol Chem 2000 : Differential effect of Rac and Cdc42 on p38 kinase activity and cell cycle progression of nonadherent primary mouse fibroblasts
Versteeg et al., J Biol Chem 2000 : In turn Rac mediates p38 mitogen activated protein ( MAP ) kinase activation and cytoskeletal reorganization, whereas factor VIIa induced p42/p44 MAP kinase stimulation required PI3K enzymatic activity but was not inhibited by dominant negative Rac proteins
Honda et al., Genes Cells 2003 : Cdc42, and presumably Rac , activated in this way, induced the activation of c-Jun N-terminal kinase (JNK), but not p38 mitogen activated protein ( MAP ) kinase or extracellular signal regulated kinase ( ERK )
Fessler et al., J Biol Chem 2004 : Methyl-beta-cyclodextrin induces activation of p38 and Cdc42, but not Rac , in the nonstimulated PMN, yet inhibits subsequent lipopolysaccharide induced activation of p38 and Cdc42
Natal et al., Apoptosis 2006 : In turn, Rac mediates the increase of both JNK and p38 activities in a sustained fashion, as well as the phosphorylation levels of their respective substrates c-Jun and ATF-2
Schreiber et al., Innate Immun 2013 : Furthermore, inhibiting Rac in HRV exposed macrophages attenuated activation of the stress kinase p38 and release of the pro-inflammatory cytokine CCL2, but inhibiting Rac did not affect release of the pro-inflammatory cytokine CCL5