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CDH1 — TWIST1

Text-mined interactions from Literome

Huber et al., Curr Opin Cell Biol 2005 (Disease Progression...) : Repression of E-cadherin by transcriptional regulators such as Snail or Twist emerges as one critical step driving EMT, and this stage is currently being molecularly linked with many of the new players
Vesuna et al., Biochem Biophys Res Commun 2008 (Breast Neoplasms) : In this study, we demonstrate that Twist can transcriptionally repress E-cadherin in breast cancer cells
Yu et al., J Cancer Res Clin Oncol 2011 (Laryngeal Neoplasms...) : Moreover, down-regulation of TWIST expression up-regulated E-cadherin and down-regulated N-cadherin expressions at mRNA and protein levels, respectively
Ng et al., J Clin Endocrinol Metab 2011 : Similarly, exogenous expression of Twist resulted in a continuous and progressive decrease in E-cadherin expression and the subsequent formation of syncytium in BeWo cells maintained under normal culture conditions
Margetts et al., Nephrol Dial Transplant 2012 : In this issue of Nephrology Dialysis Transplantation, Cuixiang Li reports on in vitro experiments where the expression of Twist led to a decreased expression of E-cadherin and the evidence of EMT
Dai et al., Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi 2012 (Carcinoma, Squamous Cell...) : E-cadherin promoter activity in response to Myc-Twist1 overexpression was measured by the dual luciferase reporter assay system ... The Twist1 significantly inhibited the E-cadherin promoter activity and enhanced the cell invasion
Cai et al., Int J Biol Macromol 2013 (Neoplasm Metastasis...) : Moreover, down-regulation of Twist expression by PGPW1 blocked epithelial-mesenchymal transition (EMT), characterized by a gain of epithelial cell markers, E-cadherin , and loss of the mesenchymal markers, vimentin and N-cadherin, at protein levels