Lorenzo-Zúñiga et al., Gut 2006
(Liver Cirrhosis, Experimental) :
IGF-I reduced intestinal permeability in BDL rats and enhanced barrier function of the monolayers of epithelial intestinal cells where lipopolysaccharide (LPS) caused a decrease in TER that was reversed by IGF-I
Schlegel et al., Histochem Cell Biol 2012
:
Application of the cell-permeable NEMO binding domain (NBD) synthetic peptide was effective to prevent NF-?B activation, but did neither block LPS induced loss of TER nor intercellular gap formation
Zhao et al., Am J Physiol Lung Cell Mol Physiol 2013
:
Inhibition of LPS mediated c-Met tyrosine ( Y1003 ) phosphorylation and internalization by prior treatment with PHA-665752, inhibition of PKCa, or overexpression of c-Met ( Y1003A ) mutant attenuated LPS induced reduction of TER