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IRF6 — NCF1

Text-mined interactions from Literome

Jinnouchi et al., J Leukoc Biol 2005 (MAP Kinase Signaling System) : Local anesthetics inhibited LPS induced up-regulation of cytochrome b558 but not LPS induced translocation of p47phox
Pacquelet et al., Biochem J 2007 : We also show that p47phox is phosphorylated in granulocytes in response to LPS stimulation ... LPS dependent phosphorylation of p47phox was enhanced by the inhibition of p38 MAPK ( mitogen activated protein kinase ), confirming that the kinase operates upstream of p38 MAPK
Yang et al., J Exp Med 2007 (Inflammation...) : This effect of LPS was mediated by the robust up-regulation of the reactive oxygen species ( ROS ) -generating nicotinamide adenine dinucleotide phosphate ( NADPH ) oxidases and the phosphorylation of p47(phox)
Qian et al., J Immunol 2007 : Mechanistic studies showed that both l- and d- morphine failed to protect dopaminergic neurons in cultures from NADPH oxidase ( PHOX ) knockout mice and significantly reduced LPS induced PHOX cytosolic subunit p47(phox) translocation to the cell membrane by inhibiting ERK phosphorylation
Wu et al., J Cell Physiol 2008 (Sepsis) : Ascorbate, apocynin and p47phox deficiency also inhibited the LPS + IFNgamma induced DNA binding activity of transcription factors IRF1 and AP1 but not NFkappaB
Qian et al., J Immunol 2008 (Inflammation) : Moreover, we demonstrate that TGF-beta1 inhibited LPS induced NADPH oxidase ( PHOX ) subunit p47phox translocation from the cytosol to the membrane in microglia within 10 min. Mechanistic studies show that TGF-beta1 fails to protect dopaminergic neurons in cultures from PHOX knockout mice, and significantly reduced LPS induced translocation of the PHOX cytosolic subunit p47phox to the cell membrane
Li et al., Arterioscler Thromb Vasc Biol 2009 (Atherosclerosis) : Furthermore, neither LPS nor DFO affected mRNA and protein levels of p47phox a nonheme containing, regulatory subunit of NADPH oxidase, or the mRNA level of NOX4, an isoform of the principal catalytic subunit of NADPH oxidase in endothelial cells
Hu et al., J Neuroimmune Pharmacol 2012 (Inflammation...) : Further mechanistic studies demonstrated that clozapine pretreatment inhibited LPS induced translocation of cytosolic subunit p47(phox) to the membrane in microglia, which was most likely through inhibiting the phosphoinositide 3-kinase (PI3K) pathway
Lee et al., Cell communication and signaling : CCS 2012 : LPS induced NADPH oxidase activation, ROS production, and p47phox translocation from the cytosol to the membrane, which were reduced by PP1 or c-Src siRNA ... We observed that LPS induced TLR4, MyD88, c-Src, and p47phox complex formation determined by co-immunoprecipitation and Western blot
Račková et al., Arch Biochem Biophys 2013 : Prolonged application of oxysterol significantly enhanced LPS stimulated association of cytosolic NADPH-oxidase factor p47[phox ] with detergent-resistant microdomains ( DRMs ) in BV-2 cells
Venaille et al., Am Rev Respir Dis 1989 (Inflammation) : Analysis of neutrophil chemotactic factor ( NCF ) production identified lung wall cells as the major source of LPS induced NCF activity in both groups and as virtually the sole source in SPF animals
Strieter et al., Science 1989 : Human endothelial cells produced a neutrophil chemotactic factor ( NCF ) upon stimulation with tumor necrosis factor-alpha (TNF-alpha), interleukin-1 beta (IL-1 beta), or lipopolysaccharide (LPS)