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JUN — TRAF3

Text-mined interactions from Literome

Urbich et al., J Clin Invest 2001 : Overexpression of TRAF-3 inhibits endothelial expression of proinflammatory cytokines and tissue factor and blocks DNA binding activity of the transcription factor AP-1 ; it thereby prevents CD40 induced endothelial activation
Lee et al., J Biochem Mol Biol 2002 : The AP1 activation by TRAF2, TRAF5 , and TRAF6 was also greatly suppressed by the dominant negative TAK1
Guasparri et al., EMBO Rep 2006 : TRAF2 and TRAF3 are required for induction of NF-kappaB and associated cell survival, as well as Jun amino-terminal kinase phosphorylation by vFLIP, whereas TRAF1, TRAF5 and TRAF6 are dispensable
Blackwell et al., Mol Cell Biol 2009 (Lymphoma) : Notably, TRAF2 phosphorylation increased both basal and inducible c-Jun and NF-kappaB activities and rendered cells resistant to stress induced apoptosis