Pathways - manually collected, often from reviews:
OpenBEL Selventa BEL large corpus:
NOXA1
→
MYC
(increases)
Evidence: downregulation of the endogenous levels of c-MYC abrogated the induction of NOXA, whereas forced expression of c-MYC conferred normal cells with the ability to accumulate NOXA and subsequently activate cell death programs in response to proteasome inhibition (Nikiforov et al., 2007).