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CRAT — JUN

Text-mined interactions from Literome

Zhou et al., Free Radic Biol Med 2001 : To test whether NF kappa B or AP-1 might be mediating the induction of GPx and CAT in muscle cells subjected to oxidative stress, we first characterized their activation by pro-oxidants
Gurney et al., J Biol Chem 1992 (Carcinoma, Hepatocellular...) : Over-expression of Jun in HepG2 cells resulted in a 10-15-fold increase in the level of transcription of a chimeric PEPCK ( -490 to +73 ) -CAT gene, while expression of Fos decreased transcription and blocked the induction of transcription from the PEPCK promoter by Jun
Touray et al., Oncogene 1991 : In NIH3T3 cells overexpression of Fos or Fos/Jun by transfection of pSV2-fos and pSV2-jun inhibited glucocorticoid dependent expression of MMTV LTR-CAT
Amato et al., Cancer Res 1998 : Consistent with these biochemical events, paclitaxel stimulated AP-1 dependent chloramphenicol acetyltransferase (CAT) reporter gene transcription in vivo, as directed from a tetradecanoyl phorbol acetate-inducible promoter