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CCL5 — NFKB1
Pathways - manually collected, often from reviews:
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Wickremasinghe et al., J Biol Chem 2004
(Tuberculosis, Pulmonary) :
Monocyte alveolar, but not upper airway, epithelial cell networks in pulmonary tuberculosis cause AP-1-, NF-IL-6-, and
NFkappaB dependent
CCL5 secretion
Nakamichi et al., J Neurochem 2005
:
Taken together, these results suggest that the recognition of dsRNA structure selectively
induces the CXCL10 and
CCL5 responses in microglia through vacuolar pH-dependent activation of
NF-kappaB and MAPK signaling pathways
Law et al., J Virol 2007
:
Our results showed that the SARS coronavirus nsp-1
plays an important role in
CCL5 , CXCL10, and CCL3 expression in human lung epithelial cells via the activation of
NF-kappaB
Liu et al., J Virol 2007
:
In contrast, siRNA mediated TLR3 knockdown did not affect RSV induced
NF-kappaB binding but did
inhibit IFN-beta, IP-10,
CCL-5 , and ISG15 expression at late times of infection
Méndez-Samperio et al., Cell Immunol 2007
(Tuberculosis) :
Sphingosine kinase, phosphatidylinositol 3-kinase, Akt,
NF-kappaB , and p300 are
required for
CCL5 production in Mycobacterium bovis Bacillus Calmette-Guérin ( BCG ) -infected epithelial cells
Ohno et al., Oral Microbiol Immunol 2008
(Inflammation) :
The
NF-kappaB inhibitor
suppressed the expression of
CCL5 and MMP9, but not that of CXCL10, whereas P. gingivalis infection induced significant CCL5 expression in MyD88 ( -/- ) osteoblasts
Filer et al., Arthritis Rheum 2009
(Arthritis, Rheumatoid) :
NF-kappaB activation was
required for production of both IL-6 and
CCL5