◀ Back to TLR2
PAM — TLR2
Text-mined interactions from Literome
Redecke et al., J Immunol 2004
(Asthma) :
In this study, we show that
activation of
TLR2 by its synthetic ligand
Pam3Cys , in contrast to activation of TLR9 by immunostimulatory DNA ( ISS-ODN ), induces a prominent Th2 biased immune response
Yoshida et al., J Biol Chem 2005
(Inflammation) :
We showed that activation of
TLR2 signaling by TLR2 ligands including peptidoglycan ( PGN ), MALP-2, and
Pam3CSK4 induces activation of IKKs-IkappaBalpha and MKK3/6-p38 pathways not only by TRAF6 but also by TRAF7, a recently identified TRAF family member
Dasu et al., Atherosclerosis 2009
(Inflammation) :
Pam3CSK4 and LPS
induced TLR2 and TLR4 expression at mRNA and protein levels are inhibited by candesartan both in vitro and in vivo
Nerren et al., Vet Immunol Immunopathol 2010
(Poultry Diseases) :
The aim of the present study was to gain better insight into the nature of the ligand for TLR15 by characterizing gene expression patterns of
TLR15 by heterophils in
response to numerous bacterial derived TLR agonists LPS, flagellin, CpG oligodeoxynucleotides, lipotechoic acid (LTA), peptidoglycan ( PGN ), and
Pam3CSK4 (PAM) , stimulation with live Salmonella enterica serovar Enteritidis ( SE-used as a positive control ), chicken isolates of Escherichia coli ( EC ) and Enterococcus gallinarum ( EG ), the equine-specific pathogen Rhodococcus equi, and stimulation with heat killed, and formalin killed SE, EC, and EG
Li et al., J Neuroimmunol 2011
(Chronic Disease...) :
Our data have shown that
stimulation of
TLR2 by TLR2 ligands peptidoglycan ( PGN ) or
Pam3CSK4 ( Pam3 ) attenuates stress induced reduction in lymphocyte numbers
Lim et al., J Immunother 2012
(Neoplasms) :
Combined
TLR Stimulation With
Pam3Cys and Poly I: C Enhances Flt3-Ligand Dendritic Cell Activation for Tumor Immunotherapy
Liu et al., Mol Cell Biochem 2013
(MAP Kinase Signaling System) :
The aim of our study is to investigate the role of RP105 in mouse macrophages
activation of TLR4 and
TLR2 signaling by lipopolysaccharides (LPS) and
Pam3CysSerLys4 ( Pam3CSK4 ) alone or in combination, and the interaction between TLR2 and TLR4 signaling through RP105