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CD36 — EPHB2
Text-mined interactions from Literome
Shen et al., FEBS Lett 2001
:
Biochemical analyses correlated
Erk activation by
Grb2-FAT with its stimulation of cell cycle progression
Cai et al., Arterioscler Thromb Vasc Biol 2004
(MAP Kinase Signaling System) :
Furthermore, an
ERK1/2 inhibitor
blocked monocyte
CD36 upregulation
Raney et al., Metabolism 2007
:
These results suggest that during moderate-intensity muscle contraction,
ERK1/2 phosphorylation is
required for translocation of
CD36 to the plasma membrane and the subsequent increase in FA uptake
Lin et al., Cardiovasc Res 2012
(Atherosclerosis) :
We showed that the inhibition of phosphatidylinositol 3-kinase (PI3K)/Akt upstream of ERK decreased SR-A and CD36 expression ; however, the inhibition of
ERK or PKCß downstream of ERK
attenuated SR-A but not
CD36 expression
Li et al., Atherosclerosis 2013
:
The specific
CD36 signal induced by oxLig-1
initiated the activation of Fyn, Lyn, caveolin-1, JNK2 and
ERK1/2 , and resulted in the up-regulation of ABCA1