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JUN — MYD88

Text-mined interactions from Literome

Chandrasekar et al., J Biol Chem 2005 : Src kinase inhibitors PP1 and PP2, phosphatidylinositol 3-kinase (PI3K) inhibitors wortmannin and LY294002, Akt inhibitor, the c-Jun N-terminal kinase (JNK) inhibitor SP600125, antisense JNK and dominant negative MyD88 , interleukin-1 receptor associated kinase ( IRAK)-1, IRAK4, and phosphatidylinositol 3-kinase expression all attenuated IL-18 mediated AP-1 binding and reporter activity, CXCL16 promoter-reporter activity, and CXCL16 expression
Ben Mkaddem et al., Cell Death Differ 2010 (Reperfusion Injury) : In this study we show that TLR4 mediates MyD88 dependent activation of TNF receptor associated factor 2, apoptosis signal regulating kinase 1 ( ASK1 ), and Jun N-terminal kinase (JNK) and p38 MAP kinases in ischemic reperfused kidneys and posthypoxic renal tubule epithelial cells ( RTECs )
Burns et al., J Biol Chem 1998 : Overexpression of MyD88 induces activation of the c-Jun N-terminal kinase (JNK) and the transcription factor NF-kappaB through its DD