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BAX — CDK2
Text-mined interactions from Literome
Morris et al., J Neurosci 2001
:
However, either p53 deficiency or the inhibition of
CDK activity alone
inhibited Bax translocation, cytochrome c release, and caspase-3-like activation
Choi et al., Apoptosis 2007
:
Thus, we suggest that
Cdk2 activity is
involved in the mitochondrial translocation of
Bax , which plays an important role in the mitochondrial membrane permeability transition during apoptotic progression
Wang et al., J Asian Nat Prod Res 2009
:
1 in high concentrations
induced Bax , caspase-3, and cyclin dependent kinase (CDK) inhibitor P21 expression, whereas the expressions of cyclin
CDK2 , Bcl-2, and proliferating cell nuclear antigen ( PCNA ) were decreased by 1 treatment
Gil-Gómez et al., EMBO J 1998
:
We found that
Cdk2 activation during thymocyte apoptosis can be
regulated by p53,
Bax and Bcl-2