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CDCA5 — EPHB2

Text-mined interactions from Literome

Harada et al., Nat Cell Biol 2001 : Furthermore, the transcription factor Egr1, is induced by NGF through the ERK pathway and mediates induction of p35 by ERK
Goodridge et al., Immunology 2003 (MAP Kinase Signaling System) : Thus, whilst LPS induced p38 mitogen activated protein kinase ( MAPkinase ) activation is required for the induction of both p40 and p35 subunits, Erk MAPkinase signalling mediates negative feedback regulation of p40, but not p35 , production
Lee et al., J Neurochem 2004 (Neuroblastoma) : Furthermore, Cdk5 and p35 expression was decreased by ERK1/2 inhibition with PD98059 and increased by overexpression of a constitutive active mitogen activated protein kinase kinase 1 ( MEK1 ) mutant, suggesting the critical role of ERK1/2 in the induction of Cdk5 and p35
Dent et al., Hepatology 2005 : Deoxycholic acid ( DCA ), chenodeoxycholic acid ( CDCA ) , taurodeoxycholic acid ( TDCA ), glycodeoxycholic acid ( GDCA ), taurochenodeoxycholic acid ( TCDCA ), glycochenodeoxycholic acid ( GCDCA ), taurocholic acid ( TCA ), glycocholic acid (GCA), and tauroursodeoxycholic acid ( TUDCA ) all activated ERK1/2 in primary rat hepatocytes that was abolished by inhibition of ERBB1, and significantly reduced by ROS quenching agents ... Treatment of rat hepatocytes with pertussis toxin ( PTX ) did not alter ERK1/2 and AKT activation induced by DCA or CDCA but abolished pathway activations by conjugated bile acids
Yui et al., Nutr Cancer 2009 : At 200 microM, DCA and CDCA induced the phosphorylation of Akt and ERK1/2 , although these phosphorylations do not appear to be indispensable for the cytoprotection