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FGFR3 — VEGFA

Pathways - manually collected, often from reviews:

• WikiPathways Focal Adhesion-PI3K-Akt-mTOR-signaling pathway: EFNA1/FGF1/FGF11/FGF10/EFNA2/EGF/FGF12/CSF1/ANGPT4/ANGPT2/ANGPT1/VEGFA/EFNA3/EFNA4/EFNA5/FGF14/FGF19/FGF17/FGF18/FGF2/FGF3/FGF4/FGF6/FGF7/FGF8/FGF9/FIGF/HGF/IGF1/INS/INS/KITLG/VEGFC/VEGFB/PDGFB/PGF/PDGFA/NGF/PDGFC/FGF21/FGF22/PDGFD/FGF20/FGF16 → FGFR2/KDR/INSR/FGFR3/IGF1R/KIT/FGFR1/EPHA2/EGFR/CSF1R/FGFR4/FLT1/FLT4/NGFR/MET/PDGFRA/PDGFRB/TEK (activation)
  

Text-mined interactions from Literome

Amizuka et al., Bone 2004 (Hyperostosis) : Furthermore, the absence of FGFR3 in single and compound mutant mice led to decreased expression of vascular endothelial growth factor ( VEGF ) and an increase in depth of hypertrophic chondrocytes
Kimura et al., Arterioscler Thromb Vasc Biol 2007 : In the contralateral arms of subjects that received the IPC stimulus, FBF responses to ACh did not change, but levels of VEGF and circulating EPCs increased
Huang et al., Vet Immunol Immunopathol 2009 : Using primary equine pulmonary artery endothelial cells culture and real-time RT-PCR method, we observed that ACh , nicotine, and muscarine inhibit the expression of E-selectin and vascular endothelial growth factor by endothelial cells stimulated by reIL-4
Yu et al., Eur Heart J 2013 (Myocardial Infarction) : In cultured cardiac microvascular endothelial cells, ACh stimulated the expression of VEGF , phosphorylation of VEGF receptor 2, and tube formation in a manner dependent upon a7-nAChR