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AR — GSK3B
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Salas et al., J Biol Chem 2004
(Prostatic Neoplasms) :
Conversely, the glycogen synthase kinase-3 beta inhibitor lithium chloride suppressed the
glycogen synthase kinase-3 beta mediated phosphorylation of the
androgen receptor , thereby enabling androgen receptor-driven transcription to occur ... Because glycogen synthase kinase-3 beta activity is suppressed after the enzyme is phosphorylated by protein kinase B/Akt and Akt activity frequently increases during the progression of prostate cancer, nullification of the
glycogen synthase kinase-3 beta mediated suppression of
androgen receptor activity by Akt likely contributes to prostate cancer progression
Liao et al., Endocrinology 2004
(Prostatic Neoplasms) :
However, the phosphatidylinositol 3kinase or
GSK-3beta inhibitors did not
block R1881 induced nuclear translocation of
androgen receptor