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IL15 — IL2RA
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Eberl et al., Cell Immunol 2002
:
HMB-PP stimulated Vgamma9/Vdelta2 T cells proliferated, expressed CCL5/RANTES, and upregulated markers like CD16,
CD25 , CD69, and CD94, in the
presence of either
IL-15 or IL-21
Lin et al., Transpl Immunol 2006
(Graft vs Host Disease) :
We found that
IL-15 differentially
enhanced CD69 and
CD25 expression on CB T cells following allo-stimulation
Zhao et al., Leukemia 2008
(Hypersensitivity, Delayed) :
sorafenib also caused G ( 0 ) /G ( 1 ) phase arrest,
inhibition of
CD25 and CD69 expression,
interleukin-2 production and LCK phosphorylation in the T cells ; all of these effects exhibited dose and time dependence
Chaput et al., Gut 2009
(Adenocarcinoma...) :
Finally,
interleukin 6 and TGF beta 1 synergistically
induced the generation of CD8 ( + )
CD25 ( + ) Foxp3 ( + ) T cells ex vivo
Ebert et al., Immunology 2009
:
IL-21 had no effect on Fas ligand (FL)- or tumour necrosis factor-alpha (TNF-alpha) mediated cytotoxicity, but it down-regulated
IL-15 stimulated expression of
CD25 and CD94, indicating that it has both positive and negative actions
Saghafi et al., Iran J Allergy Asthma Immunol 2010
(Necrosis) :
We established that
CD25 expression on CB MNCs could be
increased with
IL-15 , in 72-hour cultures, but to a lesser degree compared to that on corresponding adult PB MNCs
Shirakawa et al., Mol Cell Biol 1989
:
Identical results were obtained with the human natural killer-like cell line YT, which can be induced to express the
interleukin-2 receptor alpha subunit in
response to
interleukin-1 , cyclic AMP, or phorbol 12-myristate 13-acetate