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Katsube et al., J Leukoc Biol 2008 : The studies with pharmacological inhibitors suggest that calpain inhibition mediated neutrophil migration is mediated by activation of MEK/ERK , p38, JNK, PI-3K/Akt, and Rac
van der Hoeven et al., Biochem Pharmacol 2010 : CP-532,903 also inhibited fMLP induced F-actin formation, a downstream effector function of Rac relevant to neutrophil migration , but not activation of ERK1/2 or p38
Liu et al., Nat Immunol 2012 : The extracellular signal regulated kinase Erk potentiated activity of the G protein coupled receptor kinase GRK2 and inhibited neutrophil migration , whereas the MAPK p38 acted as a noncanonical GRK that phosphorylated the formyl peptide receptor FPR1 and facilitated neutrophil migration by blocking GRK2 function