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Jang et al., Blood Cells Mol Dis 2002
(Leukemia...) :
Furthermore, overexpression of FGFR2AT-I resulted in increased AKT and MAPK activation, conferring a survival advantage
Morales et al., Int J Biochem Cell Biol 2012
(Fibrosis) :
These results strongly suggest that the fibrotic response to Ang-II is mediated by the AT-1 receptor and requires the p38MAPK phosphorylation, NOX induced ROS, and TGF-ß1 expression increase mediated by Ang-II in skeletal muscle cells