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IRF6 — LIF

Text-mined interactions from Literome

Jensen et al., Environmental health : a global access science source 2003 : RPAs indicated that AhR+ bone marrow stromal cells consistently up-regulated genes encoding IL-6 and LIF in response to LPS , presumably through activation of Toll-like receptor 4
Anegon et al., J Immunol 1991 : Nuclear run-on analysis did not detect an increase in HILDA/LIF gene transcription upon stimulation with LPS or phorbol ester in combination with 1,25-dihydroxyvitamin D3
Sánchez-Lemus et al., Brain Behav Immun 2009 (Inflammation) : The ARB reduced LPS induced pituitary gene expression of IL-6, LIF , iNOS, COX-2 and IkappaB-alpha ; and prevented LPS induced increase of nNOS/eNOS activity
Hartner et al., Kidney Int 1994 : Strong induction of LIF mRNA expression was caused by cytokines, such as interleukin-1 beta ( 5 ng/ml ), tumor necrosis factor alpha ( 100 ng/ml ) and PDGF ( 100 ng/ml ), as well as LPS ( 200 ng/ml )
Ulich et al., Am J Physiol 1994 (Acute Disease...) : In conclusion, intratracheal LPS initiates the secretion of endogenous LIF into the alveolar space where LIF may contribute to the downregulation of LPS initiated acute neutrophilic inflammation by downregulating expression of TNF
Aloisi et al., J Immunol 1994 : LIF synthesis was significantly increased by the cytokines IL-1 beta, TNF-alpha, and TGF-beta 1, but not by IFN-gamma, IL-6, or LPS
Wang et al., Endocrinology 1996 : Hypothalamic and pituitary LIF and LIF-R are significantly induced ( up to 6- and 4-fold, respectively ) in vivo in response to lipopolysaccharide endotoxin (LPS) administered to B6D2F1 and C57BL/6 mice ... In contrast to the nearly exclusive expression of matrix associated LIF messenger RNA ( mRNA ) in control hypothalamus and pituitary, both diffusible and matrix associated LIF mRNA alternate transcripts are induced by LPS