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OSM — SOAT1

Text-mined interactions from Literome

Halfter et al., Brain Res Mol Brain Res 2000 (Glioblastoma...) : Activation of the Jak-Stat- and MAPK-pathways by oncostatin M is not sufficient to cause growth inhibition of human glioma cells
Schaefer et al., Cytokine 2000 : We report that of four gp 130 cytokines tested, only oncostatin M (OnM) resulted in the activation of Stat molecules
Tiffen et al., Mol Endocrinol 2008 : In vitro, OSM but not LIF persistently down-regulated phosphorylated ( p ) -STAT5 , even in the continued presence of prolactin
Baker et al., Glia 2010 : OSM regulates gene expression primarily by activating the JAK/STAT , NF-kappaB, and/or MAPK pathways, in a cell-type specific manner
Migita et al., Arthritis Res Ther 2011 (Arthritis, Rheumatoid) : CP690,550 pretreatment completely abrogated the OSM induced production of IL-6, as well as OSM induced JAK/STAT , and activation of mitogen activated kinases ( MAPKs ) in FLS
Korzus et al., J Biol Chem 1997 : The mitogen activated protein kinase and JAK-STAT signaling pathways are required for an oncostatin M-responsive element mediated activation of matrix metalloproteinase 1 gene expression