◀ Back to VCAM1

SOD3 — VCAM1

Text-mined interactions from Literome

Li et al., Arterioscler Thromb Vasc Biol 2002 (Hypertension) : The present study was undertaken to determine whether gene transfer of endothelial NO synthase (eNOS) or manganese superoxide dismutase ( MnSOD ) affects VCAM-1 levels in arteries from hypertensive rats
Chen et al., J Pharmacol Exp Ther 2003 : In addition, expression of superoxide dismutase by adenovirus suppressed TNF-alpha induced VCAM-1 , E-selectin, and ICAM-1 mRNA accumulation
Lin et al., Arterioscler Thromb Vasc Biol 2005 (Arteriosclerosis...) : SOD expression significantly suppressed TNF-alpha induced expression of vascular cell adhesion molecule-1 and intercellular cell adhesion molecule-1 and reduced the binding of the human neutrophils to TNF-alpha stimulated HAECs
Basta et al., Arterioscler Thromb Vasc Biol 2005 (Atherosclerosis) : The inhibition of Cu/Zn superoxide dismutase inhibited both ROS and VCAM-1 induction, indicating that H2O2 by this source is involved as a mediator of VCAM-1 expression by AGEs
Rajapakse et al., Am J Physiol Heart Circ Physiol 2009 (Hyperglycemia...) : In the cells cultured under a low-glucose condition when no increased HBP flux occurred, azaserine enhanced the manganese-superoxide dismutase ( MnSOD ) protein level and also inhibited the oxidative stress and the expression of VCAM-1 and ICAM-1 in response to TNF-alpha
Kong et al., Lipids in health and disease 2013 : However, comparing to the model rabbits, levels of TNF-a, IL-1ß and MDA decreased significantly and serum SOD activity increased , gene and protein expressions of E-sel, ICAM-1, VCAM-1 and MCP-1 in aortas decreased significantly with the treatment of kaempferol