◀ Back to FAS

FAS — IL1B

Text-mined interactions from Literome

Houghton et al., Infect Immun 2000 (Helicobacter Infections) : Of various inflammatory cytokines tested, only interleukin 1beta and tumor necrosis factor alpha induced Fas Ag expression, and removal of either of these from the conditioned medium abrogated the response
Matsumura et al., Clin Exp Rheumatol 2000 (Lymphadenitis...) : In contrast, IL-1 beta , IL-2, IL-4, and GM-CSF had no effect on Fas levels
Tanaka et al., J Reprod Immunol 2000 : Flow cytometric analyses demonstrated that TGF-beta1, IL-1beta , and EGF did not induce Fas expression on the cell surface
Rescigno et al., J Exp Med 2000 (Inflammation) : Fas engagement induces the maturation of dendritic cells (DCs), the release of interleukin (IL)-1beta , and the production of interferon gamma in the absence of IL-12 during DC-T cell cognate interaction : a new role for Fas ligand in inflammatory responses
Darville et al., Diabetes 2001 (Insulinoma...) : Inactivation of two adjacent NF-kappaB and C/EBP sites in this region abolished IL-1beta induced Fas promoter activity in RINm5F cells
Redd et al., Histochem J 2002 (Diabetes Mellitus, Type 1) : Interleukin-1beta in intra-islet macrophages may induce Fas and inducible nitric oxide synthase expression in an autocrine and paracrine manner and mediate beta cell destruction or even death of some macrophages and T cells
Wachlin et al., J Autoimmun 2003 (Diabetes Mellitus, Type 1) : However, IL-1beta induced Fas expression was significantly enhanced only on beta-cells from diabetes-prone BB rats
DosReis et al., Curr Drug Targets Inflamm Allergy 2003 (Lung Diseases...) : Fas-Fas ligand induced apoptosis promotes IL-1beta secretion, neutrophil extravasation, and loss of epithelial cells
Augstein et al., Endocrinology 2004 (Diabetes Mellitus, Type 1...) : The effect of FasL on NIT-1 cells was evident during and after the induction of Fas expression by IL-1beta and IFNgamma
Elouil et al., Diabetologia 2005 (MAP Kinase Signaling System) : IL-1beta consistently increased islet NFkappaB activity and c-Myc, haeme-oxygenase 1, inducible nitric oxide synthase (iNOS), Fas , and inhibitor of NFkappaB alpha ( IkappaBalpha ) mRNA levels
Nolsøe et al., Genes Immun 2006 (Diabetes Mellitus, Type 2...) : High glucose concentrations induce IL-1beta production in human beta-cells, Fas expression and concomitant apoptosis owing to a constitutive expression of FasL
Carmona et al., J Immunol 2006 : We additionally determined that IL-1beta and TNF-alpha secretion by beta-glucan stimulated DCs was partially regulated by Fas-FasL
Maedler et al., Diabetes 2006 (Glucose Intolerance) : In contrast, Fas induction by IL-1beta was monophasic
Augstein et al., Diab Vasc Dis Res 2008 : Induction of Fas by IL-1beta/IFN-gamma coupled with activation by Super-FasL augmented cytokine induced cell death
Kondo et al., Immunology 2009 (Myositis) : In vitro culturing of muscle cells with the proinflammatory cytokines interferon-gamma, tumour necrosis factor-alpha, and interleukin (IL)-1beta synergistically increased Fas expression, susceptibility to Fas mediated apoptosis, and the expression of cytoplasmic caspases 8 and 3
Bai-Feng et al., Diabetes Res Clin Pract 2010 : The expression of iNOS and the promoting apoptosis gene Bax and Fas were significantly up-regulated by the induction of IL-1beta and TNF-alpha
Stassi et al., J Exp Med 1997 (Diabetes Mellitus, Type 1) : NG-monomethyl-L-arginine, an inhibitor of nitric oxide ( NO ) synthase, prevents IL-1beta induced Fas expression, whereas the NO donors sodium nitroprusside and nitric oxide releasing compound ( NOC ) -18, induce functional Fas expression in normal pancreatic beta cells
Kotani et al., Rinsho Byori 1997 (Thyroiditis, Autoimmune) : Fas expression was induced by IL-1 beta stimulation
Kawakami et al., J Lab Clin Med 1998 : About 30 % of unstimulated PBLs expressed Fas , and the expression was augmented by interleukin-1beta (IL-1beta) , IL-2, tumor necrosis factor-alpha (TNF-alpha), interferon-gamma (IFN-gamma), or PMA plus ionomycin