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ANG — GPI

Text-mined interactions from Literome

Musa-Aziz et al., J Membr Biol 2005 (Colonic Neoplasms) : The effect of ANG II on pH(i) , [ Ca ( 2+ ) ] ( i ) and cell volume was investigated in T84 cells, a cell line originated from colon epithelium, using the probes BCECF-AM, Fluo 4-AM and acridine orange, respectively
Magness et al., Am J Physiol 1992 : During ovine pregnancy ANG II increases uterine PGI2 production
Carraro-Lacroix et al., Pflugers Arch 2006 : The effects of ANG on pH(i) and [ Ca(+2) ] ( i ) were assessed by the fluorescent probes, 2',7-bis ( 2-carboxyethyl ) -5 ( 6 ) -carboxyfluorescein-acetoxy-methyl ester and fluo-4-acetoxy-methyl ester, in the absence of Na ( + ) to block the Na ( + ) /H ( + ) exchanger
Nakagawa et al., J Cardiovasc Pharmacol 1987 : The enhancement of PGI2 generation induced by ANG I or BK was not affected by the pretreatment with captopril ... In rat aortic rings, ANG I, ANG II, and BK enhanced whereas captopril ( 10 ( -3 ) M ) attenuated PGI2 generation ... These results suggest that ( a ) the conversion of ANG I to ANG II did not enhance PGI2 generation in EC ; ( b ) the ANG II stimulated PGI2 generation of aortic rings was partially derived by mechanical stimulation of EC, probably originating from the contraction of smooth muscle cells by ANG II ; ( c ) captopril directly inhibited PGI2 generation ; and ( d ) in EC, the stimulation of PGI2 generation by ANG I or BK is probably regulated by an activating effect on ACE as an autoregulatory mechanism
Saito et al., Life Sci 1988 : The interaction of prostaglandin ( PG ) with the vascular renin-angiotensin (R-A) system was examined by studies on the effects of PGI2 , PGE2 and the inhibitor of PG synthesis, indomethacin, on the release of angiotensin II (Ang II) from isolated rat mesenteric arteries
Miyamori et al., Clin Exp Hypertens A 1985 (Body Weight...) : PGI2 generation which was measured by 6-keto-PGF1 alpha output in the perfusate was significantly reduced in DX treated rats at prehypertensive stage both under the basal conditions and in response to angiotensin (ANG) II, whereas in DOCA group rats, vascular PGI2 production was not impaired
Essig et al., Nephron 1997 : Ang II stimulated the production of PGI ( 2 ), but did not modify that of ET-1
Skolnick et al., Am J Physiol 1998 (Myocardial Infarction) : Effect of ANG II on pHi , [Ca2+ ] i, and contraction in rabbit ventricular myocytes from infarcted hearts