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CXCL11 — STAT3
Text-mined interactions from Literome
Burger et al., Oncogene 2005
(Cell Transformation, Neoplastic...) :
In CXCR2 transfected NIH 3T3 cells and human microvascular endothelial cells ( HMEC ), which express the CXCR2 constitutively,
STAT3 is phosphorylated upon
stimulation with IL-8 (
CXCL8 )
Yang et al., J Immunol 2007
:
Although the IFN stimulated gene ISG15 was induced to a similar extent in Daudi and DRST3 cells, expression of wild-type
STAT3 in DRST3 cells
restored the IFN inducibility of
CXCL11 ... These data indicate that
CXCL11 gene induction by IFN is
STAT3 dependent , but that phosphorylation of Y705 of STAT3 is not required
Kanda et al., Endocrinology 2011
(Psoriasis) :
This indicates the involvement of NF-?B in CXCL8, CXCL10, and CCL20 induction by visfatin and the
involvement of
STAT3 in
CXCL8 induction
Pini et al., J Leukoc Biol 2012
(Inflammation...) :
However, the delayed resolution of neutrophil infiltration ; sustained production of CXCL1,
CXCL2 , and CCL2 ; prolonged
activation of
STAT-3 ; and induction of MMP-7 in the pancreas, as well as heightened induction of serum amylase A of DIO mice, were blunted significantly in DIO IL-6 KO mice