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SLC2A4 — UBC

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: SLC2A4 — UBC (physical association, affinity chromatography technology) Foster et al., J Proteome Res 2006
• IRef Biogrid Interaction: SLC2A4 — UBC (physical association, affinity chromatography technology) Shewan et al., J Cell Sci 2013*
• IRef Biogrid Interaction: SLC2A4 — UBC (physical association, affinity chromatography technology) Lamb et al., Traffic 2010*

Text-mined interactions from Literome

Shibata et al., Rinsho Byori 2005 : Overexpression of Ubc9 resulted in inhibition of GLUT4 degradation and promotion of its targeting to the non-endosomal, non-TGN GLUT4 storage vesicles (GSV), leading to an up-regulation of GLUT4 expression level and insulin-responsive glucose transport
Liu et al., Diabetes 2007 : Overexpression of Ubc9 resulted in an inhibition of GLUT4 degradation and promoted its targeting to the unique insulin-responsive GLUT4 storage compartment (GSC) , leading to an increase in GLUT4 amount and insulin-responsive glucose transport in 3T3-L1 adipocytes