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GFAP — IL6

Text-mined interactions from Literome

Haggiag et al., J Neurosci Res 2001 : Glial fibrillary acidic protein , present in nmSC, is not significantly induced by IL6RIL6
Takanaga et al., J Biol Chem 2004 (Brain Neoplasms...) : In addition, recombinant IL-6 induced GFAP expression and STAT3 phosphorylation
DeLeo et al., Eur J Pain 1997 : Immunohistochemistry was used to examine spinal localization of the cytokines, interleukin (IL)-6 , IL-10 and glial activation ( OX-42 for microglia and anti-glial fibrillary acidic protein for astrocytes )
Takanaga et al., J Neurochem 2004 (Glioma) : Most importantly, the inhibitory effect of betaNF on GFAP promoter activity was recovered by the addition of recombinant IL-6
Woiciechowsky et al., Med Sci Monit 2004 (Gliosis) : IL-6 infusion, as well as IL-beta induced IL-6 release into the CSF, increase GFAP expression in the cerebral cortex and hippocampus
Alexander et al., Brain Behav Immun 2007 (Amyotrophic Lateral Sclerosis...) : The most common pattern was found in 50 % ( 11 out of 22 ) of the CRPS patients and consisted of ; elevated IL-6 , low levels of IL-4 or IL-10, increased GFAP or MCP1 and increases in at least two of the following markers NO metabolites, calcium or glutamate
Lee et al., J Neurochem 2009 (Wallerian Degeneration) : Interleukin-6 is required for the early induction of glial fibrillary acidic protein in Schwann cells during Wallerian degeneration ... The IL-6/STAT3 signaling in primary SCs and RT4 cells induced the gene expression of glial fibrillary acidic protein (GFAP) , which is known to be required for the proper regeneration of the injured nerves ... These findings indicate that IL-6 plays an important role in STAT3 dependent GFAP induction in SCs during peripheral nerve regeneration
Miller et al., Brain Res 2010 : Unexpectedly, there were no effects of IL-6 deficiency on GFAP , MDA or SNAP25 levels in females, but IL-6 deficiency was associated with lower cerebellar MBP ( p < 0.05 ) levels
Kahn et al., Glia 1994 : Using Northern and Western analyses in conjunction with immunocytochemistry we have demonstrated that ciliary neurotrophic factor (CNTF), leukemia inhibitory factor (LIF), and interleukin-6 (IL-6) cause a transient increase in glial fibrillary acidic protein (GFAP) in oligodendrocyte type-2 astrocyte ( O-2A ) progenitor cells ... At maximal cytokine concentrations, the largest increase in GFAP protein levels were observed for CNTF and LIF ; albeit, IL-6 did increase GFAP but the order of magnitude was 6-7 times less