◀ Back to MYLIP

ALK — MYLIP

Text-mined interactions from Literome

Wang et al., Blood 2008 : Here we report that miR-24 regulates erythroid differentiation by influencing the expression of human activin type I receptor ALK4 ( hALK4 )
Ye et al., J Cell Sci 2011 (Neoplasms, Glandular and Epithelial...) : Taken together, these results demonstrate that the Nodal-ALK7 pathway is involved in cisplatin induced cell death in ovarian cancer cells and that miR-376c enhances proliferation, survival and chemoresistance by targeting, at least in part, ALK7
Dejean et al., Leukemia 2011 (Anoxia...) : First, we observed that ALK expression leads to downregulation of miR-16 and increased Vascular Endothelial Growth Factor ( VEGF ) levels
Vishwamitra et al., Am J Pathol 2012 (Carcinoma, Non-Small-Cell Lung...) : We tested the hypothesis that the expression of ALK could be regulated by miR
Siragam et al., Oncotarget 2012 (Breast Neoplasms...) : Interestingly, miR-98 reduced the expression of ALK4 and MMP11, both of which were potential targets of miR-98
Fu et al., Hypertension 2013 (Pre-Eclampsia) : These results demonstrate that miR-376c inhibits both ALK5 and ALK7 expression to impair transforming growth factor-ß/Nodal signaling, leading to increases in cell proliferation and invasion