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SMAD3 — TGFBR3

Pathways - manually collected, often from reviews:

• WikiPathways Extracellular vesicle-mediated signaling in recipient cells: TGFBR1/TGFBR3 → SMAD4/SMAD2/SMAD3 (activation)
• WikiPathways Hypothesized Pathways in Pathogenesis of Cardiovascular Disease: TGFBR3 → Complex of TGFBR2-TGFBR1-SMAD2-SMAD3 (activation)
  

Text-mined interactions from Literome

Verschueren et al., J Biol Chem 1999 : Activation of transforming growth factor beta receptors causes the phosphorylation and nuclear translocation of Smad proteins, which then participate in the regulation of expression of target genes
Chin et al., Plast Reconstr Surg 2001 (Keloid) : Differential expression of transforming growth factor-beta receptors I and II and activation of Smad 3 in keloid fibroblasts
Luo et al., Mol Cell Proteomics 2006 : Endogenous transforming growth factor-beta receptor mediated Smad signaling complexes analyzed by mass spectrometry