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CD79A — JUN

Text-mined interactions from Literome

Perrotti et al., Mol Cell Biol 2000 : FUS proteolysis is induced by c-Jun , is suppressed by BCR-ABL or Jun kinase 1, and does not depend on c-Jun transactivation potential, ubiquitination, or its interaction with Jun kinase 1
Inabe et al., FEBS Lett 2002 (Agammaglobulinemia) : Recent studies have shown that Btk plays an important role in BCR mediated c-Jun NH ( 2 ) -terminal kinase ( JNK ) 1 activation ; however, the mechanism by which Btk participates in the JNK1 response remains elusive
Mancini et al., Traffic 2009 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : In particular, constitutive tyrosine kinase ( TK ) activity of p210 BCR-ABL blocks c-Jun N-terminal kinase (JNK) phosphorylation leading to 14-3-3 sigma phosphorylation at a critical residue ( Ser ( 186 ) ) for c-ABL binding in response to DNA damage