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BCL2 — VDAC2
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Sugiyama et al., Oncogene 2002
(Ion Channel Gating) :
We have previously shown that the
voltage dependent anion channel ( VDAC ) plays a crucial role in apoptotic changes of the mitochondria and its activity is directly
regulated by some
Bcl-2 family members, including Bcl-2/Bcl-x ( L ) and Bax/Bak but not Bid
Wei et al., Cell Signal 2009
:
In contrast,
Bcl-XS ( Ser106Ala )
induces a significant loss of
VDAC expression, and cytochrome c- and caspase independent toxicity in the non-neuronal HEK293A cells