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IL4 — RNF19A

Text-mined interactions from Literome

Schafer et al., J Immunol 1999 : Inhibitors of p38 also blocked CD4+ T cell production of IL-4 ( SB 203580 IC50 = 20-100 nM ), but not IL-2, in response to CD3 and CD28 stimulation
Nagano et al., Int Immunol 2002 : Taken together, these results suggest that both ERK and p38 ( MAPK ) pathways are involved in LPS induced COX-2 expression and PGE ( 2 ) production in neutrophils, and IL-10 and IL-4 inhibit neutrophil prostanoid synthesis by down regulating the activation of p38 ( MAPK )
Pesu et al., J Biol Chem 2002 : IL-4 stimulation did not stimulate p38 MAPK activity, but inhibition of p38 MAPK activity directly correlated with inhibition of IL-4 induced gene activation
Feito et al., Eur J Immunol 2003 (MAP Kinase Signaling System) : ERK or JNKinhibitors partially inhibited IL-4 and IL-10 secretion, while PKC or p38 inhibitors had no significant effects on IL-4 or IL-10 secretion
El Mabrouk et al., J Cell Biochem 2008 : IL-4 did not affect OSM stimulated phosphorylation of extracellular signal regulated kinases ( ERKs ), protein 38 (p38) , c-Jun N-terminal kinase (JNK) and Stat1
Chiu et al., Pediatr Neonatol 2008 : Osthol did not suppress IL-4 induced p38 , ERK or JNK expression
Ding et al., Journal of hematopathology 2009 : The differential IL-4 regulation of p38 MAPK expression in cell lines derived from DLBCL may relate to the cellular origin of these neoplasms
Liu et al., J Huazhong Univ Sci Technolog Med Sci 2010 (Rhinitis, Allergic, Seasonal) : To investigate the effect of p38 mitogen activated protein kinase ( MAPK ) on the production of Th2 cytokines such as IL-4 and IL-5 in allergic rhinitis, a model of allergic rhinitis was established in SD rats