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IRF6 — TNFSF13B
Text-mined interactions from Literome
Scapini et al., Blood 2005
(Arthritis, Rheumatoid) :
We show that CXCL1/GROalpha, CXCL8/IL-8, C5a, immune complexes, tumor necrosis factor-alpha (TNF-alpha), leukotriene B4, N-formyl-methionyl-leucyl-phenylalanine ( fMLP ), and
lipopolysaccharide (LPS) , which by themselves do not
induce BLyS de novo synthesis, act as potent secretagogues for BLyS, which is mainly stored in Golgi related compartments within G-CSF treated neutrophils, as determined by immunogold electron microscopy
Moon et al., Free Radic Biol Med 2006
:
BAFF expression was
augmented by
LPS stimulation and by serum deprivation that induced ROS production
Alsaleh et al., Arthritis Rheum 2007
(Arthritis, Rheumatoid) :
In contrast to IFNgamma, neither TNFalpha,
LPS , BLP, nor CpG
induced the de novo synthesis and release of
BAFF by FLS
Chu et al., J Immunol 2007
:
Stimulation of B2 and B1 cells with
LPS or CpG-oligodeoxynucleotides
induced MyD88 dependent plasma cell differentiation and intracellular expression of
BAFF and APRIL
Moon et al., Int Immunopharmacol 2011
(Lupus Erythematosus, Systemic) :
Chemicals also inhibited
LPS stimulated
BAFF production from splenocytes
Moon et al., Exp Cell Res 2011
:
Lipopolysaccharide (LPS) increased
BAFF expression through the activation of toll-like receptor 4 (TLR4) dependent signal transduction