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CCNG1 — PCNA
Text-mined interactions from Literome
Bouchard et al., J Virol 2001
:
Instead, HBx is shown to activate the cyclin A promoter,
induce cyclin A-cyclin dependent kinase 2 complexes, and promote
cycling of growth arrested cells into G1 through a pathway involving activation of Src tyrosine kinases
Piscopo et al., Cancer Res 2008
:
Ectopic
cyclin G1 protein stability is
increased by cyclin box mutation or by association with inactive
cyclin dependent kinase (CDK) subunits, suggesting that a function of cyclin G1 as a CDK regulator may be required for its rapid turnover
Santo et al., Oncogene 2010
(Multiple Myeloma) :
Dysregulated cell
cycling is a universal hallmark of cancer and is often
mediated by abnormal activation of
cyclin dependent kinases (CDKs) and their cyclin partners
Soffar et al., Int J Radiat Biol 2013
(Head and Neck Neoplasms) :
Abstract Purpose
: Cyclin dependent kinase 2 (CDK2) is critically
involved in cell
cycling and has been proposed as a potential cancer target
Elias et al., Int J Dermatol 1995
(Psoriasis) :
Since
PCNA is a marker of cellular proliferation and p53
inhibits cellular
cycling , some of the beneficial effects of PTU and MMI in psoriasis may depend on the ability of the drugs to impair cellular turnover, perhaps by binding to the triiodothyronine ( T3 ) receptor