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SOAT1 — TYK2

Text-mined interactions from Literome

Shang et al., Cancer Sci 2007 (Carcinoma, Renal Cell...) : The results suggest that the resistance of RCC to IFN-alpha is associated with the lack of Jak1, Tyk2 and Stat1 expression and defective Jak-Stat activation , but not with a lack of IFN-alpha receptor, suppressors of cytokine signaling induction or other factors examined
Shimoda et al., Blood 1997 (Fibrosarcoma) : In the absence of Jak1, G-CSF induces activation of Jak2 and Tyk2 , but fails to induce receptor tyrosine phosphorylation and induces dramatically reduced levels of Stat activation